Commonly co-occurs w/ rib fractures, flail chest, hemothorax, or pneumothorax.
Most common potentially lethal chest injury in children/young adults.
Clinical Features
Onset: Rapidly develops within 24 hours of trauma (often <15 mins to 6 hrs).
Sx: Dyspnea, tachypnea, pleuritic chest pain, hemoptysis (rare but possible).
PE: Localized chest wall ecchymosis, chest wall tenderness, decreased breath sounds, and crackles over the injured lung segment.
Diagnosis
Initial: CXR showing patchy, non-segmental alveolar infiltrates/consolidation (described as “patchy opacification”) not restricted by anatomical lobar borders.
Note: CXR may be normal in the first 6 hours; serial CXRs or CT indicated if clinical suspicion is high.
Confirmatory/Most Sensitive: Chest CT (reveals patchy ground-glass opacities or consolidation immediately after trauma).
Key Labs/Monitoring:
ABG: Hypoxemia (PaO2 < 60 mmHg or SaO2 < 90% on room air), hypocapnia (due to hyperventilation).
Continuous pulse oximetry.
Differential Diagnostics
ARDS: Diff by bilateral infiltrates, delayed onset (typically 24–72 hours post-insult), and systemic inflammatory trigger (e.g., sepsis, pancreatitis). Pulmonary contusion is localized to the trauma site and presents <24 hours.
Hemothorax: Diff by dullness to percussion, decreased breath sounds, and CXR showing pleural fluid/layering rather than parenchymal infiltrates.
Aspiration Pneumonitis: Diff by history of witnessed aspiration/emesis, altered mental status, and infiltrates localized to dependent lung segments (e.g., RLL).
Myocardial Contusion: Diff by signs of cardiogenic shock, elevated troponins, and ECG abnormalities (e.g., new BBB, arrhythmia).
Management
Initial/Supportive:
Supplemental O2 (maintain SaO2 > 92%).
Aggressive pain control (systemic analgesics or epidural/intercostal nerve blocks) to prevent splinting, atelectasis, and hypoventilation.
Fluid Optimization: Judicious IVF administration. Avoid volume overload as injured capillaries are hyperpermeable, which can worsen pulmonary edema.
Refractory/Severe:
Non-invasive positive pressure ventilation (NIPPV/CPAP) for moderate hypoxemia.
Intubation w/ mechanical ventilation + low-tidal-volume (VT) ventilation + PEEP if refractory hypoxemia, severe flail chest, or respiratory failure occurs.
Imaging: CT Chest (shows airway disruption, “windsock sign”, persistent air outline outside the tracheobronchial tree).
Confirmatory/Gold Standard: Bronchoscopy (directly visualizes the location, size, and extent of the airway tear).
Differential Diagnostics
Simple/Tension Pneumothorax: Diff by rapid lung re-expansion and cessation of air leak after chest tube insertion (tracheobronchial injury has persistent massive air leak and failure of lung to re-expand). c
Esophageal Rupture: Diff by presence of hydropneumothorax, pleural fluid analysis (↑ amylase, low pH), and contrast esophagography/CT w/ oral contrast (no persistent large-volume air leak).
Diaphragmatic Rupture: Diff by CXR showing abdominal contents (stomach/bowel loops) in the hemithorax, w/ NG tube tip positioned in the chest.
Management
Immediate Airway Stabilization (ABC):
Secure airway via bronchoscopy-guided intubation (advancing ET tube distal to the site of injury/tear to prevent positive-pressure ventilation from worsening the pneumomediastinum/PTX).
Place large-bore chest tubes (often multiple required due to massive air leak).
Surgical Intervention (Definitive):
Emergent operative primary repair (thoracotomy) for large tears (>1/3 airway circumference), persistent major air leak, respiratory failure, or associated esophageal/vascular injury.
Conservative Management:
Nonoperative observation for stable pts w/ small (<2 cm or <1/3 circumference) lesions, minimal air leak, and fully expanded lung on chest tube suction.
Complications
Mediastinitis (high mortality risk).
Bronchial/tracheal stenosis (leading to atelectasis and dyspnea weeks/months later).