Epidemiology


Etiology

  • Population: Military recruits, competitive runners, ballet dancers, gymnasts.
  • Mechanisms & Triggers:
    • Abrupt ↑ in training volume, frequency, or intensity without adequate recovery.
    • Repetitive mechanical overload exceeding bone remodeling capacity.
  • Common Sites:
    • 2nd and 3rd metatarsal shafts (most common overall; “march fracture”).
    • Posteromedial tibial shaft (most common lower leg). c
    • Fibula, calcaneus, navicular, femoral neck.
  • Risk Factors:
    • Female Athlete Triad: Low energy availability/eating disorder, amenorrhea/oligomenorrhea, ↓ BMD (osteopenia/osteoporosis).
    • Nutritional deficiencies: Low Vit D, inadequate Ca intake, anorexia nervosa
      • Consider these if happens in male c
    • Biomechanical abnormalities: Cavus feet (high arch), leg-length discrepancy.

Tip

The female athlete triad syndrome is associated with an increased risk of stress fractures.


Pathophysiology


Clinical features

  • History:
    • Insidious onset of localized, aching pain.
    • Initially occurs only with weight-bearing or end of training session; relieved by rest.
    • Progresses to pain during daily ambulation, and eventually rest/night pain.
  • Physical Exam:
    • Focal point tenderness directly over the affected bone cortex (hallmark). c
    • Localized soft tissue edema and erythema.
    • Positive “tuning fork” or percussion test (vibration reproduces sharp, localized pain).
    • Antalgic gait.

Diagnostics

  • X-ray: recommended initial imaging study
    • Radiographic features of stress fractures include:
      • Fracture line: line of faint lucency
      • Indirect features: periosteal thickening, increased cortical density, formation of a callus, endosteal thickening and/or sclerosis
    • Often normal in the first 2–3 weeks of disease onset

Differential Diagnostics

  • Medial Tibial Stress Syndrome (“Shin Splints”):
    • Diff by diffuse tenderness along posteromedial tibial border (>5 cm) without focal point tenderness; XR normal; MRI shows periosteal edema without cortical line or deep marrow edema.

Treatment

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