Epidemiology


Etiology


  • A transient syndrome of acute brain dysfunction, not a specific disease.
  • Results from a wide range of underlying medical conditions, substance intoxication/withdrawal, or medications.
  • Key Pathophysiology: Believed to involve widespread disturbances in brain metabolism, neurotransmitter function (especially ↓ acetylcholine, ↑ dopamine), and inflammation.

Risk factors

  • Advanced age
  • Neurologic disorder (e.g. dementia, stroke)
  • Sensory impairment (e.g. hearing loss)
  • Sleep disruption
  • Immobilization

Precipitating causes

Pathophysiology


Clinical features


  • Acute onset (hours to days) and a fluctuating course are hallmarks.
  • Core feature: Disturbance in attention and awareness.
  • Associated features include disorganized thinking, altered sleep-wake cycle, perceptual disturbances (visual hallucinations are common), and psychomotor changes.
  • Can be hyperactive (agitation), hypoactive (lethargy, more common and often missed), or mixed.
  • Typically occurs in elderly, hospitalized, or post-operative patients, especially those with pre-existing cognitive impairment like dementia.

Diagnostics


  • Differential diagnostics
    • Mania: cognition is intact; not disoriented; treated with lithium

Treatment


  • Polypharmacy/psychoactive drug avoidance
  • Sleep facilitation: bright day/dim night lighting
  • Visual/hearing aids
  • Mobilization: out of bed, restraint avoidance

Treatment of agitation in delirium

  • Agitation should initially be managed with nonpharmacologic strategies.
    • Avoid physical restraints as much as possible in older patients with delirium, as they can worsen distress and agitation, as well as contribute to preventable injuries.
  • Pharmacotherapy

Warning