FeatureHeat CrampsHeat ExhaustionHeat Stroke
Core ProblemElectrolyte lossWater & salt lossThermoregulatory failure
Core TempNormal< 40°C (104°F)> 40°C (104°F)
CNS StatusNormal / AlertIntact (dizzy, headache)Altered (confusion, seizure, coma)
SkinMoistCool, pale, moistHot, dry OR moist
Tx PriorityElectrolyte fluidsCool down, fluidsImmediate rapid cooling (e.g., ice bath)

Epidemiology & Risk Factors

  • Exertional Heat Stroke (EHS):
    • Young, healthy individuals (athletes, military recruits, outdoor laborers).
    • Strenuous physical exertion in hot/humid environments with inadequate acclimation.
  • Non-Exertional / Classic Heat Stroke (NEHS):
    • Extremes of age (elderly, infants), poor socioeconomic status (lack of air conditioning during heatwaves), chronic medical illness (CVD, psychiatric illness).
    • Medications impairing thermoregulation:
      • Anticholinergics / Antihistamines / TCAs: Inhibit diaphoresis.
      • Diuretics: Dehydration and volume depletion.
      • Beta-blockers: Blunt compensatory cutaneous vasodilation and cardiac output.
      • Sympathomimetics / Amphetamines / Cocaine: ↑ Endogenous heat production.
      • Alcohol: Promotes peripheral vasodilation and diuresis.

Clinical Features

  • Heat Cramps:
    • Painful, involuntary muscle contractions (usually calves, thighs, shoulders) during/after heavy exercise.
    • Pathophysiology: Dilutional hyponatremia from rehydrating with large amounts of free water without electrolytes.
  • Heat Exhaustion:
    • Core body temperature < 40°C (104°F).
    • Systemic symptoms: Headache, nausea, vomiting, dizziness, tachycardia, profuse diaphoresis, malaise.
    • Intact CNS function (no significant encephalopathy, delirium, or coma).
  • Heat Stroke (Medical Emergency):
    • Core body temperature > 40°C (104°F) + CNS dysfunction (AMS, confusion, ataxia, seizures, delirium, coma).
    • EHS: Profuse sweating often still present initially; rapid onset.
    • NEHS: Skin typically anhidrotic (hot, dry); insidious onset over days.

Diagnosis

  • Initial/Screening:
    • Rectal core temperature (essential; oral/tympanic/axillary probes significantly underestimate true core temp).
    • Continuous cardiorespiratory monitoring, pulse oximetry, and blood glucose check.
  • Confirmatory:
    • Clinical diagnosis based on rectal core temp > 40°C (104°F) + CNS dysfunction in the setting of heat exposure.
  • Key Labs:
    • BMP: Hypokalemia or hyperkalemia, hyponatremia/hypernatremia, elevated BUN/Cr (prerenal azotemia or ATN).
    • Serum CK & Urine Myoglobin: Marked elevation in EHS (indicates rhabdomyolysis).
    • LFTs: AST/ALT markedly elevated (frequently in the thousands; acute hepatic necrosis).
    • Coagulation Panel: Prolonged PT/INR, PTT, low platelets, elevated D-dimer, low fibrinogen (DIC). c
    • ABG/VBG: Lactic acidosis, initial respiratory alkalosis progressing to mixed metabolic acidosis.
  • Imaging & ECG:
    • ECG: Sinus tachycardia, conduction abnormalities, QT prolongation, or signs of hyperkalemia.
    • Chest X-ray: Rule out noncardiogenic pulmonary edema (ARDS) or aspiration.
    • CT Head / Lumbar Puncture: Indicated only if CNS dysfunction persists post-cooling to rule out alternative intracranial pathology or meningitis.

Differential Diagnostics

  • Heat Exhaustion vs. Heat Stroke:
    • Diff by core temp < 40°C and normal mental status in heat exhaustion.
  • Neuroleptic Malignant Syndrome (NMS):
    • Diff by history of dopamine antagonists (e.g., haloperidol, fluphenazine), “lead-pipe” muscle rigidity, hyporeflexia, and leukocytosis.
  • Serotonin Syndrome (SS):
    • Diff by history of serotonergic agents (SSRIs, SNRIs, MAOIs, tramadol), hyperreflexia, neuromuscular clonus (ocular/ankle), and prominent GI symptoms (diarrhea).
  • Malignant Hyperthermia (MH):
    • Diff by intraoperative or immediate postoperative onset following volatile halogenated inhalational anesthetics (e.g., sevoflurane) or succinylcholine; severe generalized rigidity and hypercapnia.
  • Thyroid Storm:
    • Diff by signs of hyperthyroidism (goiter, exophthalmos, high lid lag, atrial fibrillation), suppressed TSH, elevated free T4/T3.
  • Anticholinergic Toxicity:
    • Diff by classic toxidrome: “Blind as a bat, mad as a hatter, red as a beet, hot as a hare, dry as a bone, full as a flask” (mydriasis, decreased bowel sounds, acute urinary retention).
  • Sepsis / Meningitis:
    • Diff by infectious source, meningeal signs (nuchal rigidity, Kernig/Brudzinski), and CSF pleocytosis.

Management

  • Immediate Priorities (ABCDE):
    • Airway protection (intubation if GCS ≤ 8 or active status epilepticus), high-flow supplemental oxygen, aggressive IV access.
  • Rapid Cooling (Target: Core temp < 39°C / 102.2°F within 30 minutes; discontinue cooling at 38.5°C to avoid rebound hypothermia):
    • Exertional Heat Stroke (EHS):
      • First-line: Ice water immersion (cold water immersion bath is gold standard).
      • Second-line / Alternative: Evaporative/convective cooling if immersion tub is unavailable.
    • Non-Exertional Heat Stroke (NEHS):
      • First-line: Evaporative and convective cooling (continuous misting with lukewarm water [20–25°C] combined with large electric fans).
      • Note: Ice water immersion is poorly tolerated and associated with higher mortality in elderly patients due to severe shivering, peripheral vasoconstriction, and precipitation of arrhythmias.
  • Fluid Resuscitation & Supportive Care:
    • Moderate room-temperature/cold IV crystalloids (0.9% Normal Saline or Ringer’s Lactate); avoid over-aggressive boluses in NEHS to prevent pulmonary edema.
    • Shivering control: IV Benzodiazepines (e.g., Lorazepam, Diazepam) to prevent secondary endogenous heat production and rhabdomyolysis.
    • Contraindication: Antipyretics (Acetaminophen, Aspirin) are ineffective (hypothalamic set point is normal) and contraindicated due to worsened hepatotoxicity and bleeding diathesis.
  • Refractory / Invasive Cooling:
    • Cold peritoneal lavage, gastric lavage, or extracorporeal life support (ECMO) in severe refractory cases.

Complications

  • Rhabdomyolysis with myoglobinuria leading to Acute Tubular Necrosis (ATN) / AKI.
  • Disseminated Intravascular Coagulation (DIC) due to thermal endothelial damage and systemic inflammatory response.
  • Acute Liver Failure / Ischemic & Heat-Induced Hepatic Necrosis.
  • Acute Respiratory Distress Syndrome (ARDS).
  • Cerebral Edema / Permanent Cerebellar Damage (Purkinje cells are hypersensitive to hyperthermia).
  • Multi-Organ Dysfunction Syndrome (MODS) and cardiovascular collapse.