- Classifications
- By Location:
- Small Bowel Obstruction (SBO): Blockage in the small intestine. More common.
- Large Bowel Obstruction (LBO): Blockage in the large intestine.
- By Severity:
- Partial: Allows some liquid and gas to pass. May resolve with conservative Tx.
- Complete: Allows nothing to pass. Usually requires surgical intervention.
- By Pathophysiology:
- Mechanical: A physical barrier obstructing the lumen (e.g., adhesion, tumor). The most common type.
- Functional (Ileus): Impaired peristalsis without a physical barrier.
- By Vascular Compromise:
- Simple: Obstruction without vascular compromise.
- Strangulated: Obstruction where blood flow is compromised, leading to ischemia and necrosis. A surgical emergency.
- Closed-Loop: A segment of bowel is obstructed at two points (e.g., volvulus or LBO with a competent ileocecal valve). High risk of rapid strangulation.
- Etiology
- SBO: Most common causes are Adhesions (from prior surgery), Bulges (hernias), and Cancer (malignancy).
- LBO: Most frequent causes are malignancy (colorectal cancer), volvulus (sigmoid and cecal), and diverticular disease.
- Functional Obstruction (Ileus): Often occurs post-operatively or due to medications (e.g., opioids), electrolyte imbalances (especially hypokalemia), and severe illness.
- Pathophysiology
- A mechanical blockage leads to the accumulation of gas and fluid proximal to the obstruction, causing bowel distention.
- Increased intraluminal pressure can compromise blood flow to the bowel wall, leading to ischemia, necrosis, and perforation.
- Significant fluid shifts into the bowel lumen (“third-spacing”) and vomiting can cause severe dehydration and electrolyte abnormalities.
- Clinical Presentation
- Cardinal symptoms include colicky abdominal pain, nausea/vomiting, abdominal distention, and obstipation (inability to pass flatus or stool).
- SBO: Pain is often mid-abdominal and colicky. Vomiting is typically early and bilious in proximal obstructions, becoming feculent in distal obstructions.
- LBO: Presentation is often more insidious, with more pronounced distention and less vomiting, especially if the ileocecal valve is competent.
- Physical Exam: Early on, bowel sounds may be high-pitched and hyperactive (“tinkling”), but they become hypoactive or absent later, especially with ischemia. Peritoneal signs (e.g., rigidity, rebound tenderness) suggest perforation or strangulation.
- Diagnosis
- Abdominal X-ray (AXR): Often the initial imaging study.
- SBO: Shows multiple dilated loops of small bowel (>3 cm) with air-fluid levels, often in a “step-ladder” pattern.
- LBO: Shows marked colonic dilation (>6 cm, >9 cm for cecum) proximal to the obstruction.
- CT Scan (Abdomen/Pelvis with contrast): This is the gold standard imaging modality. It can confirm the diagnosis, identify the location and cause of the obstruction (the “transition point”), and detect complications like ischemia, perforation, or a closed loop.
- Labs: A complete blood count (CBC) may show leukocytosis (suggesting ischemia). An elevated lactate level is a red flag for ischemia. Electrolyte panels often reveal dehydration and disturbances (e.g., hypokalemic, hypochloremic metabolic alkalosis from vomiting).
- Management
- Initial/Conservative (“Drip and Suck”):
- NPO (Nil Per Os - nothing by mouth).
- IV fluid resuscitation to correct dehydration and electrolyte imbalances.
- Nasogastric (NG) tube decompression to remove fluid and air from the stomach and proximal bowel, relieving distention and vomiting.
- This approach is often successful for partial SBO due to adhesions.
- Surgical Intervention:
- Indications: Complete or closed-loop obstructions, signs of bowel compromise (ischemia, necrosis, perforation), strangulated hernias, or failure of conservative management (typically after 48 hours).
- The specific procedure depends on the cause (e.g., lysis of adhesions, hernia repair, bowel resection).
- Complications
- Bowel ischemia and necrosis (strangulation).
- Perforation, leading to peritonitis and sepsis.
- Dehydration and acute kidney injury.
- Electrolyte imbalances.